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A considerable portion of the preclinical literature involves the administration of semaglutide prior to or concurrently with disease induction, thereby reflecting a prevention or very early intervention paradigm rather than a treatment approach

This enzyme achieves this promiscuity primarily through rapid transpersulfidation of C201 in a CstB-unique loop relative to a canonical PDO, followed by molecular shuttling of a covalently bound sulfonate group to a persulfidated C408, and thiosulfate release